MY BATTLE AGAINST CANCER: Survivor protocol : foreword by Thomas Seyfried

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Halestrap, A.P. The Monocarboxylate Transporter Family—Structure and Functional Characterization. IUMB Life 2012, 64, 1–9. [ Google Scholar] [ CrossRef] He’s had no traditional treatment, but simply did lots of fasting and taking supplements and he’s been diagnosed as cancer free since last June. His metastasis are gone and only the old scars remain on his bone scans. This first article in the starve cancer series is quite instructive. I had no idea that glucose levels could be brought down to the levels reported in the article. I have seen the tumor responses to fairly modest reduction in glucose in mice models; one can only guess what might happen if glucose levels were brought down to the extent imagined in the article. Bonuccelli, G.; Tsirigos, A.; Whitaker-Menez, D.; Pavlides, S.; Pestell, R.G.; Chiavarina, B.; Frank, P.G.; Flomenberg, N.; Howell, A.; Martinez-Outschoorn, U.E.; et al. Ketones and lactate “fuel” tumor growth and metastasis. Cell Cycle 2010, 9, 3506–3514. [ Google Scholar] [ PubMed]

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As I watched the Pca spread in scans get worse, the number of bone mets has increased, and I have many little ones not making PsMa hence Lu177 or Ac225 won'k, but Ra may work, but mean extension of lifetime of Ra223 is 4 months, but then men seeking Ra223 have tried all other things and have very high Psa, maybe 600, or 10 times what mine is. Israël, M. Genetic adaptation controlled by methylations and acetylation’s at the nuclear and cytosolic Levels: A hypothetical model. Neurochem. Res. 2003, 25, 631–635. [ Google Scholar] [ CrossRef] Wilson, B.R.; Bogdan, A.R.; Miyazawa, M.; Hashimoto, K. Siderophores in iron metabolism: From Mechanism to therapy. Trends Mol. Med. 2016, 22, 1077–1090. [ Google Scholar] [ CrossRef][ Green Version] Aslam, M.N.; Bergin, I.; Naik, M.; Hampton, A.; Allen, R.; Kunkel, S.L.; Rush, H.; Varani, J. A multi-mineral natural product inhibits liver tumor formation in C57BL mice. Biol. Trace Elem. Res. 2012, 147, 267–274. [ Google Scholar] [ CrossRef][ Green Version]Fiorillo, M.; Peiris-Pagès, M.; Sanchez-Alvarez, R.; Bartella, L.; Di Donna, L.; Dolce, V.; Sindona, G.; Sotgia, F.; Cappello, A.R.; Lissanti, M.P. Bergamot natural products eradicate cancer stem cells (CSCs) by targeting mevalonate, Rho-GDI-Signaling and mitochondria metabolism. BBA-Bioenerg. 2018, 1859, 984–996. [ Google Scholar] [ CrossRef] An early observation by Brünings, recalled by Klement [ 27], deserves to be mentioned. In his study, patients were given a low-carbohydrate diet, which was associated with an insulin injection protocol that aimed to decrease the supply of glucose to tumors. Initially he observed a decrease in tumor sizes, and later a rebound, while feeding patients with a high-fat diet. We now know that insulin elicits the release of somatostatin from delta cells, which decreases glucagon and ketogenesis leading to a probable decrease in BHB and growth of ketone-dependent tumors. However, since Brünings fed patients with a high-fat ketogenic diet, a rebound of tumors took place. Brünings was not aware that somatostatin release triggered by the insulin injections was probably behind the initial reduction in tumors [ 28]. Indeed, somatostatin decreases glucagon and ketogenesis at the beginning of the trial; however, a late rebound of tumors probably occurs when insulin fails to maintain somatostatin release. In time, the decline in somatostatin stops inhibiting glucagon, which then boosts ketogenesis, providing ketones to the tumor [ 29]. In an earlier work on animal cancer models, we observed that octreotide, a somatostatin analog, decreases tumor volumes [ 12]. Cassim, S.; Vučetić, M.; Ždralević, M.; Pouyssegur, J. Warburg and Beyond: The Power of Mitochondrial Metabolism to Collaborate or replace Fermentative Glycolysis in Cancer. Cancers 2020, 12, 1119. [ Google Scholar] [ CrossRef] [ PubMed] This book tells my story, the story of a carefree man leaving a happy life in Miami, who suddenly falls into the nightmare of the disease whose just name scares you: cancer. Mine is incurable, or rather all of mine because the prostate, bones and lymph nodes are affected. Despite the terrible prognosis from my oncologists, I have decided that I will not die of cancer. Do you think that's impossible? Yet I should be dead by the time of this writing. The point is, I have never felt this good in years and my latest results point to remission! I want Radium223, but docs don't really know if that will work, and there are not many add on drugs to make it work better.